ALLMAX, AMINOCORE BCAA, Watermelon, 2.1 lbs (945 g)

The Dominant Muscle Builder 

Why use branched chain amino acids (BCAA)? Supplementation is crucial for athletes seeking to build muscle. BCAA are the primary building blocks for muscle growth, reduced muscle breakdown and facilitation of fat loss. Unlike other amino acids, BCAA aminos – leucine, valine and isoleucine – cannot be produced by the body, so must be provided through diet or supplementation.

Why use ALLMAX AMINOCORE? Not all BCAA are created equal. AMINOCORE delivers 8.18g of BCAA in a 9:6:5 ratio clinically proven to provide a 350% increase in activation of muscle growth signals! The purest, free-form source of BCAA, AMINOCORE has no added fillers or non-BCAA aminos. AMINOCORE’S 9:6:5 BCAAs are 100% pure prior to flavoring. Typical 2:1:1 sources contain impurities resulting in cloudiness, oily foam, sediment, and the worst – a harsh bitter taste! AMINOCORE is the cleanest, purest, best-tasting BCAA supplement money can buy!

Intelligent diet support. AMINOCORE feeds your muscles during diet and exercise to dramatically decrease muscle loss. Additional B-Vitamins further assists in enhancing metabolic power and improving energy levels.

The science of muscle growth. AMINOCORE 9:6:5 BCAA ratio is clinically proven to increase the activation of muscle growth signals. Scientific studies have identified mTor-p70S6K as the trigger directly responsible for extreme muscle growth. Supplementing with BCAA has been shown to activate increased mTor-p70S6K cellular signaling responsible for muscle growth. The result: you build and retain more hard earned lean muscle and maintain a higher metabolism.‡

This product has been skip-lot tested for banned substances for the assurance of athletes and supplement users under the Informed Choice program.

‡Combined with intense resistance training, research has shown that 8,180 mg of BCAA in a 9:6:5 ratio increase signaling within muscle cells to upregulate protein synthesis (growth) by 350%. Research conducted at the Nobel Prize winning Karolinska Institute in Stockholm, Sweden. Journal reference: Karlsson et. al. Am J Physiol Endocrinol Metab 287: E1-E7, 2004.